linkstream2 microblog

QT:[[”
“As he grew in his profession, Six came to feel he had a right to express himself on the family collection. A series of clashes with his father ensued, many of them about providing greater public access, which has always been a difficulty. Currently, tours of the
collection, which are by appointment only, are booked into next year. The picture that the younger Six sketched was of an inward-looking father who is trying to preserve a legacy by keeping the world at bay, who comes to realize over time that he also has to do battle with a gregarious and extroverted son who feels that the way to preserve that legacy is precisely by sharing it with the wider world. The battles left the younger Six progressively more exasperated: “I would cycle home after and think, Jesus, Dad, I’m trying to help you.””
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Rembrandt in the Blood: An Obsessive Aristocrat, Rediscovered Paintings and an Art-World Feud
https://www.nytimes.com/2019/02/27/magazine/rembrandt-jan-six.html

https://www.markey.senate.gov/news/press-releases/senators-markey-and-hawley-introduce-bipartisan-legislation-to-update-childrens-online-privacy-rules

Balanced perspective on history and future of genomic medicine by Jay Shendure https://www.cell.com/cell/fulltext/S0092-8674(19)30152-7

https://www.newyorker.com/news/our-columnists/how-i-would-cover-the-college-admissions-scandal-as-a-foreign-correspondent

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3523303/

discusses NDE & NIE

https://mailchi.mp/add7f814b67d/neurodevelopments-720229
https://www.libd.org/neurodevelopments/

the Lieber’s neuroDEVELOPMENTS newsletter, which features the PsychENCODE paper

prediction markets
https://www.predictit.org/

http://cebp.aacrjournals.org/content/27/4/363.long

QT:[[”
The first GWAS on lung cancer were reported in 2008. Three independent studies identified a susceptibility locus on chromosome 15q. Hung and colleagues (14) found two SNPs strongly associated with lung cancer on chromosome 15q25. Further genotyping in this region revealed many SNPs in tight linkage disequilibrium (LD) showing evidence of association. Six genes are located in this region including three nicotinic acetylcholine receptor subunits (CHRNA5, CHRNA3, and CHRNB4). Interestingly, no appreciable variation in the risk was found across smoking categories or histologic subtypes of lung cancer. In a second GWAS, a SNP within the CHRNA3gene was strongly associated with smoking quantity and nicotine dependence (15). The same SNP was also strongly associated with lung cancer. The results suggest that the variant on chromosome 15q25 confers risk of lung cancer through its effect on tobacco addiction.
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